Subtitle Insomnia
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This book, which is written as a reader-friendly guide, is intended for clinical trainees, non-insomnia sleep specialists, and for expert CBT clinicians from outside the sleep medicine field who wish to begin the process of learning to provide empirically validated CBT-I.
The Book is organized into seven parts: definition of insomnia; review of the conceptual; framework for treatment; overview of the components of therapy; session-by-session guide; dialogues; assessment and eligibility for CBT-I; and sample documentation. The organizing principles for the guide can best be expressed as two seemingly simple questions:
Dr. Perlis is an Associate Professor of Psychiatry and URMC Neurosciences Program at the University of Rochester and is the Director of the UR Sleep Research Laboratory. His clinical expertise is in the area of Behavioral Sleep Medicine. His research interests include sleep in psychiatric disorders and neurocognitive phenomena in insomnia, the mechanisms of action of sedative hypnotics, and the development of alternative treatments for insomnia. He was a founding member of the AASM's Behavioral Sleep Medicine Committee and of the editorial board for the Journal of Behavioral Sleep Medicine. He currently serves on the editorial boards of the Journal of Sleep Research and the Journal SLEEP and he hosts an annual seminar on CBT-I for those interested in learning the techniques.
Harvey and Tang suggested increased cortical arousal, abnormal neuronal circuitry, and frequent night awakenings as etiological factors for paradoxical insomnia (10). Additionally, in a study evaluating the EEG frequency spectra, increased arousal during sleep was observed in PI patients (11). However, the difficulty in maintaining sleep in patients diagnosed with OSA can also be explained by the stimulation that occurs during sleep (12). Frequent arousal caused by increased respiratory events in patients with OSA can mimic the repetitive arousal that occurs in PI, thus causing the patient to confuse sleep and wakefulness. This situation may explain the association of OUA and PI. However, our patient did not receive any treatment for OSA except for a change in position.
Disturbances of sleep are commonly seen in many of the DSM-5 psychiatric disorders. Furthermore, psychiatric symptoms are commonly experienced in association with sleep disorders. This chapter reviews some basic physiology of sleep-wake regulation as well as the most common sleep disorders of importance to the practicing psychiatrist. Included are insomnia, restless legs syndrome, obstructive sleep apnea, narcolepsy, idiopathic and other hypersomnias, and parasomnias such as sleepwalking/sleep terrors and REM sleep behavior disorder. In each case, diagnostic criteria are described, based both upon the American Psychiatric Association Diagnostic and Statistical Manual, Fifth Edition, and the International Classification of Sleep Disorders, Second Edition (ICSD-2). Discussions of epidemiology, clinical features, typical case examples, laboratory findings, course, differential diagnosis, etiology, and treatment considerations will enable the reader to recognize these disorders in their patients and to facilitate their treatment.
N2 - Disturbances of sleep are commonly seen in many of the DSM-5 psychiatric disorders. Furthermore, psychiatric symptoms are commonly experienced in association with sleep disorders. This chapter reviews some basic physiology of sleep-wake regulation as well as the most common sleep disorders of importance to the practicing psychiatrist. Included are insomnia, restless legs syndrome, obstructive sleep apnea, narcolepsy, idiopathic and other hypersomnias, and parasomnias such as sleepwalking/sleep terrors and REM sleep behavior disorder. In each case, diagnostic criteria are described, based both upon the American Psychiatric Association Diagnostic and Statistical Manual, Fifth Edition, and the International Classification of Sleep Disorders, Second Edition (ICSD-2). Discussions of epidemiology, clinical features, typical case examples, laboratory findings, course, differential diagnosis, etiology, and treatment considerations will enable the reader to recognize these disorders in their patients and to facilitate their treatment.
AB - Disturbances of sleep are commonly seen in many of the DSM-5 psychiatric disorders. Furthermore, psychiatric symptoms are commonly experienced in association with sleep disorders. This chapter reviews some basic physiology of sleep-wake regulation as well as the most common sleep disorders of importance to the practicing psychiatrist. Included are insomnia, restless legs syndrome, obstructive sleep apnea, narcolepsy, idiopathic and other hypersomnias, and parasomnias such as sleepwalking/sleep terrors and REM sleep behavior disorder. In each case, diagnostic criteria are described, based both upon the American Psychiatric Association Diagnostic and Statistical Manual, Fifth Edition, and the International Classification of Sleep Disorders, Second Edition (ICSD-2). Discussions of epidemiology, clinical features, typical case examples, laboratory findings, course, differential diagnosis, etiology, and treatment considerations will enable the reader to recognize these disorders in their patients and to facilitate their treatment.
The introduction gives an overview of the research findings in the area of study, focusing on the variables the authors are considering, i.e. the influence of dark personality traits, perceived stress and moral judgments on insomnia.
Objectives: Although the six hypotheses were listed, only the second objective was mentioned, but not the first. Page 2, lines 75-76 is described the second objective \"So, the second aim of this study was to investigate the relationship between personality characteristics, perceived stress, and insomnia.\" The first aim was described in the abstract. Usually the objectives are mentioned and then the hypotheses.
Results: From Table 04 which shows the results of the Multiple linear regression it does not appear that the interaction between the variables was tested. The results of this table indicate that both psychopathy and ethical judgments predict insomnia, but not the interaction of these variables as described in rows 199 and 200, as well as in the abstract rows 20 and 21.
Martin Scorsese. USA, 1976. Original language, Spanish subtitles. 113'. To deal with the chronic insomnia he's suffered since his return from Vietnam, Travis Bickle decides to work nights driving a taxi. He has little contact with people in his private life, but he notices the violence and desolation swallowing up New York City. Travis starts writing his impressions in his diary, until one day he decides to take action.
A large number of studies have shown a close association between the 24-hour rhythms in core body temperature and sleep propensity. More recently, studies have have begun to elucidate an intriguing association of sleep with skin temperature as well. The present chapter addresses the association of sleep and alertness with skin temperature. It discusses whether the association could reflect common underlying drivers of both sleep propensity and skin vasodilation; whether it could reflect efferents of sleep-regulating brain circuits to thermoregulatory circuits; and whether skin temperature could provide afferent input to sleep-regulating brain circuits. Sleep regulation and concomitant changes in skin temperature are systematically discussed and three parallel factors suggested: a circadian clock mechanism, a homeostatic hourglass mechanism, and a third set of sleep-permissive and wake-promoting factors that gate the effectiveness of signals from the clock and hourglass in the actual induction of sleep or maintenance of alert wakefulness. The chapter moreover discusses how the association between skin temperature and arousal can change with sleep deprivation and insomnia. Finally it addresses whether the promising laboratory findings on the effects of skin temperature manipulations on vigilance can be applied to improve sleep in everyday life.
N2 - A large number of studies have shown a close association between the 24-hour rhythms in core body temperature and sleep propensity. More recently, studies have have begun to elucidate an intriguing association of sleep with skin temperature as well. The present chapter addresses the association of sleep and alertness with skin temperature. It discusses whether the association could reflect common underlying drivers of both sleep propensity and skin vasodilation; whether it could reflect efferents of sleep-regulating brain circuits to thermoregulatory circuits; and whether skin temperature could provide afferent input to sleep-regulating brain circuits. Sleep regulation and concomitant changes in skin temperature are systematically discussed and three parallel factors suggested: a circadian clock mechanism, a homeostatic hourglass mechanism, and a third set of sleep-permissive and wake-promoting factors that gate the effectiveness of signals from the clock and hourglass in the actual induction of sleep or maintenance of alert wakefulness. The chapter moreover discusses how the association between skin temperature and arousal can change with sleep deprivation and insomnia. Finally it addresses whether the promising laboratory findings on the effects of skin temperature manipulations on vigilance can be applied to improve sleep in everyday life.
AB - A large number of studies have shown a close association between the 24-hour rhythms in core body temperature and sleep propensity. More recently, studies have have begun to elucidate an intriguing association of sleep with skin temperature as well. The present chapter addresses the association of sleep and alertness with skin temperature. It discusses whether the association could reflect common underlying drivers of both sleep propensity and skin vasodilation; whether it could reflect efferents of sleep-regulating brain circuits to thermoregulatory circuits; and whether skin temperature could provide afferent input to sleep-regulating brain circuits. Sleep regulation and concomitant changes in skin temperature are systematically discussed and three parallel factors suggested: a circadian clock mechanism, a homeostatic hourglass mechanism, and a third set of sleep-permissive and wake-promoting factors that gate the effectiveness of signals from the clock and hourglass in the actual induction of sleep or maintenance of alert wakefulness. The chapter moreover discusses how the association between skin temperature and arousal can change with sleep deprivation and insomnia. Finally it addresses whether the promising laboratory findings on the effects of skin temperature manipulations on vigilance can be applied to improve sleep in everyday life. 59ce067264